KCNA3 and myocardial infarction: Even if sevoflurane offered cellular protection via different mechanisms, such as intracellular signaling pathways[30], mitochondrial function[31], and potassium channels[32], recent studies have suggested that sevoflurane does not significantly reduce mortality or major postoperative complications, such as myocardial infarction, ventilation time and intensive care unit stay[7–8].