Neutrophil‐specific NAT10 overexpression mitigates neutrophil pyroptosis and reduces sepsis‐related mortality in mice by reversing the ULK1‐STING‐NLRP3 axis.[143] Pyroptosis‐induced cell death is recognized as a factor in various diseases, including cardiovascular diseases, neurological disorders, and liver conditions. The gene discussed is NAT10; the disease is Sepsis.