For instance, binding of lncRNA OCC‐1 enhanced ubiquitination of HuR, suppressing the growth of CRC cells16; LINC0051 promoted proliferation of CRC in vitro by binding to EZH2 and enhancing its enrichment in the promoter region of IL‐2417; CYTOR bound to NCL and Sam68 as a scaffold and facilitated migration, invasion, proliferation and metastasis in CRC.18 This evidence concerns the gene CYTOR and colorectal carcinoma.