Preliminary research has also shown that the expression level of PLK1 in NKTCL is increased, while Resveratrol and icariin could inhibit the activity of PLK1 and promote the apoptosis of NKTCL tumor cells [2, 11], which suggests that PLK1 may play an important role in the occurrence and development of NKTCL. The gene discussed is PLK1; the disease is extranodal nasal NK/T cell lymphoma.