CD14 and triple-A syndrome: Another single-cell transcription profiling study on human aortic tissues revealed three monocyte clusters (CD14+, FCGR3A+, CD36+, HLA-DRA+) and macrophage clusters (CD14+, FCGR3A+, CD68+, TFRC+), indicating the upregulation of inflammatory genes involved in cytokine-mediated signaling, nuclear factor-κB transcription factor activity, antigen processing, and T lymphocyte costimulation in monocyte/macrophage populations from AAA samples18.