EVs in the serum of patients with AAA exhibited differences in miR-122-5p, miR-193a-5p, miR-543, miR-576-3p, miR-629-5p, miR-2110, and miR-483-5p, which regulate biological functions involved in cell growth, aging, neuron death, vasculature development, kinase signaling pathway, and the TGF-β response116. Here, TGFB1 is linked to triple-A syndrome.