Moreover, the complex formed by ETS1 with p52 NF-κB, whose expression is specifically controlled by non-canonical NF-κB signalling, has been shown to bind selectively the mutant TERT promoter −146A thus promoting telomerase expression in glioblastoma and melanoma (Li et al., 2015; Vallarelli et al., 2016). The gene discussed is TERT; the disease is melanoma.