CYTOR and head and neck squamous cell carcinoma: Previously, we have demonstrated that FOSL1 is the dominant AP‐1 family member in HNSCC that controls EMT, maintains cancer stemness, and promotes metastasis.[9] Thus, based on our bioinformatics analysis, we hypothesized that CYTOR may regulate FOSL1 signaling to facilitate the malignant phenotypes of HNSCC.