Apart from its predicted localization, little is known about CASC4’s functionality, in cancer or otherwise, apart from the fact that CASC4 mRNA undergoes alternative splicing [22], that shed and secreted CASC4 can induce a migratory phenotype in triple-negative breast cancer [21], and that CASC4 suppresses Tn-antigen mediated metastasis in breast cancer [23]. Here, GOLM2 is linked to breast carcinoma.