CLEC4D and tuberculosis: The upregulated proteins quantified via label-free methods following infection by either pathogenic bacterium types were primarily enriched in pathways such as the NF-kappa B signalling pathway, IL-17 signalling pathway, C-type lectin receptor signalling pathway, tuberculosis, cytokine–cytokine receptor interaction, inflammatory mediator regulation of TRP channels, Toll-like receptor signalling pathway, peroxisome, and HIF-1 signalling pathway (Figure 3D).