However, at the SOC level of congenital, familial and genetic disorders, aplasia and uridine diphosphate glucuronosyltransferase 1A1 (UGT1A1) gene mutation is the main reported PTs, which were not recorded in the specification of SG or clinical trials, and more likely to be related to the patient’s development problems or incomplete documentation of AEs information (TRODELVY). The gene discussed is UGT1A1; the disease is hereditary disease.