KMT2A and gastric cancer: Additionally, in the study, it was also demonstrated that when the nucleus translocation of β-catenin signaling was efficiently inhibited by Tegatrabetan (BC2059) treatment in gastric cancer cells, KMT2A overexpression did not significantly upregulate the levels of these stemness-related molecules and rescue the proliferation as well as inhibit the apoptosis of gastric cancer cells.