In line with this, EATL and RCD-II share several pathophysiological features, including a common genetic background (i.e. homozygosity for HLA-DQ2; common allelic variants of MYO9B gene) (52, 53) and overlapping mutations in the JAK-STAT and NF-kB pathway (57). This evidence concerns the gene SOAT1 and enteropathy-associated T-cell lymphoma.