Additional events in the pathogenesis of EATL include oncogenic mutations in TET2, POT1, DDX3X, PRDM1/BLIMP1 and KMT2D (57, 97), deletions of 16q12.1 and gains of 1q, 5q and 9q (98). The gene discussed is PRDM1; the disease is enteropathy-associated T-cell lymphoma.