Whereas, Enz treatments significantly reduced the recruitment of mTOR (or mTOR/AR) to KLK3, ENO1, SLC26A3, and TMPRSS2 in LNCaP, C4-2B, and MDA PCa 2b, but not to KLK3 and SLC26A3 in the Enz-resistant 22Rv1 (miR-99b-5p/Enz vs. NC, Figure 6). The gene discussed is KLK3; the disease is posterior cortical atrophy.