As shown by the representative images (Figures 4A–C, E–G), in comparison with the immunoreactivity of NMDAR2A and NMDAR2B in CTX, NMDAR2A and NMDAR2B immunoreactivity were intensively increased in the dysplastic neurons of FCD IIb and TSC lesions. Here, GRIN2A is linked to cerebrotendinous xanthomatosis.