Moreover, LINC01503 could facilitate cell migration, infiltration, and epithelial–mesenchymal transition in cholangiocarcinoma cells [30], whereas CYTOR was up-regulated and significantly associated with the poor prognosis of the cancer patients[31], In HNSCC, CYTOR inhibited cell apoptosis following treatment with the chemotherapeutic drug diamminedichloroplatinum (DDP) [32]. This evidence concerns the gene CYTOR and cholangiocarcinoma.