PMP22 and Charcot-Marie-Tooth disease type 1A: Human iPSC or iPSC-differentiated cells are considered to be the optimal model for genetic events underlying CMT1A, because no humanized disease genome model carrying duplicated human PMP22 genome together with long flanking regions of human genome around PMP22 gene is currently available and because Human iPSC or iPSC-differentiated cells are considered a simple system to test genetic events underlying CMT1A.