IL6 and idiopathic pulmonary fibrosis: Ultimately, through transwell co-cultures and neutrophil derivative experiments, potential mast cell and neutrophil – fibroblast interactions in the lungs via cytokines and chemokines (e.g., IL-6, IL-8) lead to the recruitment and activation of more immune cells (e.g., neutrophils) that perpetuate inflammatory mechanisms to add to pulmonary remodeling in IPF.