Both C1 and C14 had similar expression profiles and expressed PPARG, the resident alveolar macrophage marker.[2, 22] However, notably, C14 was from lung tumor with low expression of FN1, whereas C1 was from lung NAT and highly expressed FN1 (M2 marker).[23] Additionally, several negative regulators were involved in inflammation inhibition in the tumor microenvironment. This evidence concerns the gene FN1 and neoplasm.