NFKB1 and acute respiratory distress syndrome: Corroborated by western blotting results, the postulated mechanism of SERP 30 involves the inhibition of LPS-induced phosphorylation of p38 and p65 via NF-κB and MAPK signalling pathways, leading to the protection of endothelial glycocalyx in LPS induced-acute respiratory distress syndrome (ARDS) in mice.