GPX4 and anemia (phenotype): Considering that RIPK3 deletion could prevent compensated anemia in glutathione peroxidase 4 (Gpx4)‐deficient mice and BMF in Tak1mutTnfr−/− mice,[23] we investigated whether Abin1Q478H/Q478H drives hematopoiesis defects through an RIPK3‐dependent pathway.