Given that RIPK3 participates in various necroptosis‐independent signaling pathways, such as apoptosis, senescence, inflammation, and autophagy,[24] we further examined whether its deletion prevents anemia in Abin1Q478H/Q478H mice by blocking RIPK1–RIPK3–MLKL axis‐mediated necroptosis. The gene discussed is MLKL; the disease is anemia (phenotype).