The granular expression of IFNß, a major downstream target of STING, was elevated in TECs from melanoma‐bearing Atg5BECKO mice, whereas its expression was blunted in TECs of Atg5/STINGBECDKO mice and comparable to WT TECs (Fig 5A), functionally validating the TEC phenotype of the DKO mice. The gene discussed is ATG5; the disease is melanoma.