Interestingly, one study found that the Sting inhibitor C‐176 significantly attenuated neuroinflammation, thereby reducing neurological deficits in ischemic stroke mice.[11b] Li et al.[27] showed that activation of the cGas‐Sting pathway could produce microglial inflammasomes and pyroptosis in microglial cells, thereby amplifying the inflammatory response during brain ischemia/reperfusion injury. The gene discussed is STING1; the disease is ischemic stroke.