A recent study evaluating the correlates of protection against RSV in a nonhuman primate model found that upper respiratory control of viral infection was associated with virus-specific IgA levels, neutralisation and complement activity, whereas lower respiratory control was associated with Fc-mediated effector mechanisms such as NK cell-mediated antibody-dependent cellular cytotoxicity (ADCC) [74]. The gene discussed is CD79A; the disease is viral infectious disease.