For instance, inhibitors of fatty acid amide hydrolase (FAAH) and monoacylglycerol lipase (MAGL), the enzymes involved in the degradation of the endocannabinoid’s anandamide and 2-arachidonoylglycerol, respectively, were reported to have anti-hyperalgesic and anti-allodynic effects in different models of neuropathic pain [17,18,19]. This evidence concerns the gene FAAH and neuropathic pain.