SLC5A1 and cardiac arrest: Regarding SGLT1, despite its presence (low amount) in neurons of the hippocampus, reports of SGLT1 alteration associated with pathological conditions are rare; increased SGLT1 expression was described as a consequence of seizures or traumatic brain injury (TBI) in experimental models, and also in the brain from an individual that died after TBI, as compared to those who died of cardiac arrest [39].