In a study conducted by Fatima et al., type II diabetes patients expressed a significantly higher serum level of IL-1β, IL-6, IL-17A, and IL-22 levels compared to controls, suggesting that besides the involvement of Th1 and Th2 cells, Th17 cells might be involved in type II diabetes [151] pathogenesis as in psoriasis. This evidence concerns the gene IL6 and type 2 diabetes mellitus.