Moreover, the same article has shown that CD4+/FOXP3− cells presenting a mutant TET2 have a higher probability of developing a TFH phenotype, a fact that leads to the consideration that this might be one of the mechanisms that increases the likelihood of developing AITL or PTCL-NOS TFH [79]. Here, TET2 is linked to angioimmunoblastic T-cell lymphoma.