The research group highlighted that mice expressing TBL1XR1:TP63, which is the most common TP63 fusion, develop T-cell lymphomas and the fusion coordinates the recruitment of nuclear receptor corepressor (NCoR)-histone deacetylase (HDAC3) and lysine methyltransferase 2D (KMT2D), which are necessary for fusion-dependent survival. This evidence concerns the gene TP63 and T-cell non-Hodgkin lymphoma.