In addition, not all analytes were lowered in persons who used MA, including CCL11 (eotaxin), IL-9, IL-16, CXCL10 (IP10), CCL3 (MIP1α), CCL4 (MIP1β), TNF-α and TNF-β (in both MA use and dependence), and G-CSF, HGF, CCL2 (MCP1), PDGF-BB, CXCL12 (SDF-1α), IL-9, and IL-10 (in MA dependence). This evidence concerns the gene CSF3 and microtia.