PLVAP and metabolic dysfunction-associated steatotic liver disease: In addition, in the disease progression of NAFLD‐related HCC, the continuous hypoxia microenvironment induces the secretion of VEGFA by hepatocytes and activates the VEGFA‐PlVAP axis to promote angiogenesis, leading to impaired liver self‐repair function, ultimately promoting the progression of NAFLD‐related diseases.