TH-302 and anti–VEGFR-2 (αVEGFR-2) did elicit greater granulocytic myeloid-derived suppressor cell (G-MDSC) infiltration into pancreatic tumors, however, as well as increased production of the tumor cell–derived chemokine CC chemokine ligand 9 (CCL9). This evidence concerns the gene KDR and neoplasm.