This association is relevant to PCa in terms of lipid metabolism, androgen receptor status, drug resistance, proliferation, apoptosis, and the downregulation of steroid 5 alpha‐reductase 2 (SRD5A2), Snail family transcriptional repressor 2 (SNAI2), Rho family GTPase 3 (RND3), and caveolin‐1 (CAV1).15, 16. This evidence concerns the gene CAV1 and posterior cortical atrophy.