TAP2 and cervical cancer: The introduction of exogenous Wnt1 partially counteracted the negative impacts of LMP2 and TAP2 overexpression, while the inhibition of endogenous Wnt1 attenuated the tumor-promoting effects of LMP2 and TAP2 depletion, highlighting the pivotal role of the Wnt1 pathway in mediating the biological functions of LMP2 and TAP2 in cervical cancer cells.