Even though we acknowledge the limitations of overexpression experiments, in the sense that may change the cell and lead to an alteration of the cellular distribution of ATG9A, we could confirm that GFP-ATG9A strongly localized at the Golgi in mock-infected cells and that this colocalization is lost with infection (Fig 4D), as observed for endogenous ATG9 (Fig 4A). The gene discussed is ATG9A; the disease is infection.