PKD1 and spontaneous coronary artery dissection: Using 13,722 sequenced controls from UK Biobank, no genes from a candidate list (130 genes associated with arteriopathies in humans and 303 genes associated with phenotypes in mice) were found to have a clear genetic association meeting statistical threshold, with the strongest signal of enrichment found for variants in PKD1 (identified in 1.7% of SCAD cases versus in 0.1% of controls, but not meeting study-wide significance).