Though not likely regulated directly by φCbK TgrL, the transcription of a subset of oxidative stress defense genes including the catalase-peroxidase katG (CCNA_03138), alkyl hydroperoxide reductase subunits C and F (CCNA_03012 and CCNA_03013, respectively), and an AhpD-family hydroperoxidase (CCNA_03812), were markedly repressed post-infection (S3 Table) suggesting that φCbK infection represses oxidative stress response. The gene discussed is CCNA2; the disease is infection.