In type 3 MG, morphology-wise, we observed MG widen around the cell body instead of extending their already-formed filopodia, which might have led to an impaired secondary phagocytotic behavior, as proposed by Agarwal et al. Additionally, Iba-1 is involved in membrane ruffling and phagocytosis in MG [36]. The gene discussed is AIF1; the disease is myasthenia gravis.