HDAC4 and myocardial infarction: Studies have shown that inhibition of HDAC1 (91), HDAC3 (92), HDAC4 (93), HDAC5 (94), HDAC6 (95), and HDAC9 (96) has a protective effect on cardiomyocyte apoptosis, left ventricular remodeling (LVR), cardiac disfunction, and cardiac fibrosis after MI.