[16] However, it shall be emphasized that other so-called ‘non-haemostatic’ properties of platelets, may be relevant for the prevention of bleeding including receptors relevant for platelet adhesion during inflammation, e.g., platelet surface expression the collagen receptor, GPVI and angiogenetic factors from platelets that could be directly involved in the pathogenesis of HHT. Here, GP6 is linked to hereditary hemorrhagic telangiectasia.