CAV1 and glaucoma: Cav-1 hyperphosphorylation, however, has also been previously demonstrated in RGCs [84], Astrocytes [156], and neurons [157] under stress conditions while in cultured cell lines derived from the trabecular meshwork of glaucoma patients, treatment with dexamethasone, the phosphorylation level of this protein was reduced [158], suggesting that Cav-1 phosphorylation under different circumstances is mediated in a cell-specific manner and that in glaucomatous conditions it is possibly restricted to RGCs.