A previous study showed that baicalein prevented AAA formation [29], while another study determined that baicalein has also been shown to significantly attenuate Ang II-induced inflammation, oxidative stress and multiple signaling pathways (Akt/mTOR, ERK1/2, NF-κB and calcineurin) in Ang II-treated mice [36]. This evidence concerns the gene NFKB1 and triple-A syndrome.