In particular, high levels of values of cardiac troponin I (cTnI) and T (cTnT), revealing the presence of an acute myocardial injury [8], were frequently found in patients with COVID-19-related pneumonia and/or sepsis and are associated with adverse outcomes such as cardiac failure, arrhythmias, and death [6,7]. This evidence concerns the gene TNNT2 and COVID-19.