Moreover, ILG improved hepatic steatosis, induced with the high-fat diet, by suppressing the expression of hepatic lipogenesis genes (FAS and SCD1) and reducing the liver triglyceride and fatty acid accumulation (plasma glutamic oxaloacetic transaminase (GOT) and glutamic pyruvic transaminase (GPT)), and at the same time enhancing β-oxidation in the liver. Here, GPT is linked to Hepatic steatosis.