TGFBR1 and myeloid sarcoma: These findings suggest that increased expression of miR-142 isoforms (miR-142-3p and miR-142-5p) may be involved in the pathogenesis of autoimmune neuroinflammation, such as MS, influencing T cell differentiation, and this effect may be mediated by the interaction of miR-142 isoforms with SOCS1 and transforming growth factor receptor beta 1 (TGFBR-1) transcripts [95].