Our previous study showed that female Wistar-Kyoto rats with spontaneous hypertension and the RA-induced model of detrusor overactivity (DO)—diagnosed in 60–90% of patients with OAB [45]—exhibited similar changes in CGRP, OCT3, and VAChT levels, as reported in the study by Wróbel et al. (2020) [2]. Here, SLC18A3 is linked to hypertensive disorder.