Thus, elevated serum levels of H-FABP and high NLR before PCI could predict the development of AKI secondary to myocardial ischemia, while high NLR and urinary L-FABP 12 h after PCI could predict CI-AKI, and high urinary L-FABP levels and NLR 72 h after PCI could define AKI patients. This evidence concerns the gene FABP1 and acute kidney injury.