FABP1 and acute kidney injury: The detected increase in urinary L-FABP in T1 than in T0 samples indicated the effect of prerenal ischemia secondary to AMI, which induces up-regulation of the expression levels of L-FABP to bind the ischemia-induced lipid peroxidation products and transferring them to urinary spaces [36] and this might explain the reported significant difference in urinary L-FABP levels in T1 samples of AKI and No-AKI patients.