This finding suggests a novel therapeutic strategy that involves targeting ATF2 or NRF2 in combination with BETi to improve cancer treatment outcomes.49 Moreover, the study on microRNAs highlights their role in regulating Ras Suppressor Protein 1 and PINCH1, further underscoring the complexity of the molecular pathways involved in adhesion and survival signaling in breast cancer. The gene discussed is ATF2; the disease is cancer.