Shen et al. (Shen et al., 2022a) established a MI model by ligating the left anterior descending coronary artery in rats and found that salvianolic acid B activated the NRF2 signalling pathway, increased SLC7A11, GPX4, FPN, and FTH1 expression, inhibited ferroptosis, and alleviated MI. This evidence concerns the gene FTH1 and myocardial infarction.