AKT1 and central nervous system cancer: Circ‐DICER1 was reported that it could interact with miR‐103a‐3p/miR‐382‐5p to inhibit ZIC4, which accelerated Hsp90beta in glioma‐exposed endothelial cells (GECs), and Hsp90 promoted the tumorigenesis of GECs binding to PI3K/AKT signaling pathway.89