The key etiological agents responsible for the development of cervical cancer are human papillomaviruses (HPVs).411 It has been established that TEAD1 transcription factor activates the early promoter of human papillomaviruses.412 In addition, a study reported that TEAD1 mediated HPV early gene expression is regulated at the transcriptional level by VGLL1 (Vestigial-Like Family Member 1), which is a TEAD-interacting transcriptional co-activator. Here, VGLL1 is linked to cervical carcinoma.