This is consistent with its early tumorigenic role reported in the malignant transformation of B cells and epithelial cells in other EBV‐associated malignancies.[20, 42] Supportively, previous studies in NKTCL have also linked LMP1 to the survival, proliferation, and even chemoresistance of cancer cells through the activation of MAPK and NF‐κB pathways.[38, 43]. The gene discussed is NFKB1; the disease is extranodal nasal NK/T cell lymphoma.