These genes are essential components of the JAK‐STAT and NF‐κB signaling pathways, which have been implicated in the oncogenesis of NKTCL[32] and other EBV‐associated malignancies.[21, 33] Consistently, SCENIC analysis revealed upregulated TF activities in the oncogenic JAK‐STAT (STAT3 and STAT5A) and NF‐κB (NKFB1, NKFB2, and RELB) pathways in NK_C9_CXCL13 (Figure S6D, Supporting Information). Here, SOAT1 is linked to extranodal nasal NK/T cell lymphoma.